Nick Craddock

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

There is increasing evidence that genome-wide association (GWA) studies represent a powerful approach to the identification of genes involved in common human diseases. We descri...

The beginning of the end for the Kraepelinian dichotomy

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Combined Analysis from Eleven Linkage Studies of Bipolar Disorder Provides Strong Evidence of Susceptibility Loci on Chromosomes 6q and 8q

Bayesian refinement of association signals for 14 loci in 3 common diseases

Improved imputation of low-frequency and rare variants using the UK10K haplotype reference panel

Abstract Imputing genotypes from reference panels created by whole-genome sequencing (WGS) provides a cost-effective strategy for augmenting the single-nucleotide polymorphism (...

Common polygenic variation contributes to risk of schizophrenia and bipolar disorder

Genome-wide association analysis identifies 13 new risk loci for schizophrenia

Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder

Genome-wide significant associations in schizophrenia to ITIH3/4, CACNA1C and SDCCAG8, and extensive replication of associations reported by the Schizophrenia PGC

Common variants at VRK2 and TCF4 conferring risk of schizophrenia

Common sequence variants have recently joined rare structural polymorphisms as genetic factors with strong evidence for association with schizophrenia. Here we extend our previo...

Genome-Wide Association Study of Schizophrenia in a Japanese Population

Association between genetic variation in a region on chromosome 11 and schizophrenia in large samples from Europe